![]() ![]() The Dryden chloralkali plant and the nearby Dryden Pulp and Paper Company were both subsidiaries of the British multinational, Reed International.īackground Mercury poisoning An expert report in 2016 confirmed that the Wabigoon River was "still highly contaminated" and that "it can be cleaned safely". For generations, many suffered with symptoms of mercury poisoning, including Minamata disease. Grassy Narrows and Whitedog First Nation communities were severely affected. : 62 : 2207 These fish, which have been a staple food of First Nations people for generations, became unsafe to eat. : 2207Ĭlay lake, the first major lake downstream from the Dryden mill, had extremely high mercury levels in predatory fish such as the walleye. ![]() The glacial clay of the area is believed to have facilitated the bioaccumulation of mercury the ecosystem. This aquatic ecosystem had extensive mercury contamination by 1970, leading to the closure of the commercial fishery and some tourism related businesses. The Wabigoon River forms a vast river system with the English River-which includes many lakes and tributaries-and together they flow west to the Winnipeg River. It was described as "one of the worst cases of environmental poisoning in Canadian history." The contamination poisoned many people in the Grassy Narrows First Nation and Whitedog First Nation communities Mercury contamination in Grassy Narrows was an uncontrolled discharge of between 9,000 kilograms (20,000 lb) and 11,000 kilograms (24,000 lb) of mercury from the Dryden Mill's chloralkali plant in Dryden into the headwaters of the Wabigoon River in the Kenora District of Northwestern Ontario from 1962 until 1970. Gnanashanmugam G, Balakrishnan R, Somasundaram S, Parimalam N, Rajmohan P, Pranesh MB (2018) Mercury toxicity following unauthorized siddha medicine intake - a mimicker of acquired neuromyotonia - report of 32 cases.Environmental & health disaster 1962 - 1970 Zhou Z, Zhang X, Cui F, Liu R, Dong Z, Wang X, Yu S (2014) Subacute motor neuron hyperexcitability with mercury poisoning: a case series and literature review. A case with possible manganese poisoning. Haug B, Schoenle P, Karch B et al (1989) Morvan’s fibrillary chorea. Reeback J, Benton S, Swash M, Schwartz MS (1979) Penicillamine-induced neuromyotonia. Hill A, Bergin P, Hanning F, Thompson P, Findlay M, Damianovich D, McKeage MJ (2010) Detecting acute neurotoxicity during platinum chemotherapy by neurophysiological assessment of motor nerve hyperexcitability. Nicholson D, Scalettar R, Jacobs R (1986) Rheumatoid rigor: gold induced myokymia. Surana S, Kumar R, Pitt M, Hafner P, Mclellan A, Davidson J, Prabakhar P, Vincent A, Hacohen Y, Wright S (2019) Acquired neuromyotonia in children with CASPR2 and LGI1 antibodies. ![]() Liang GH, Järlebark L, Ulfendahl M, Moore EJ (2003) Mercury (Hg2+) suppression of potassium currents of outer hair cells. (2004) Features and safety of mercury in the Tibetan medicine “Zota”. Eur J Case Rep Int Med 4(6):000632Įkino S, Susa M, Ninomiya T, Imamura K, Kitamura T (2007) Minamata disease revisited: an update on the acute and chronic manifestations of methyl mercury poisoning. Malek A, Aouad K, El Khoury R et al (2017) Chronic mercury intoxication masquerading as systemic disease: a case report and review of the literature. Muscle Nerve 52(1):5–12Ĭlarkson T, Magos L, Myers G (2003) The toxicology of mercury-current exposures and clinical manifestations. Ahmed A, Simmons Z (2015) Isaacs syndrome: a review. ![]()
0 Comments
Leave a Reply. |
AuthorWrite something about yourself. No need to be fancy, just an overview. ArchivesCategories |